Author index

Acta Physiologica 2009; Volume 197, Supplement 674
Belgian Society for Fundamental and Clinical Physiology and Pharmacology, Autumn Meeting 2009
10/24/2009-10/24/2009
Free University of Brussels, Brussels, Belgium

A  |  B  |  C  |  D  |  E  |  F  |  G  |  H  |  I  |  J  |  K  |  L  |  M  |  N  |  O  |  P  |  Q  |  R  |  S  |  T  |  U  |  V  |  W  |  X  |  Y  |  Z D De Bock M. - Connexin channels contribute to endothelial Ca2+ dynamics and alter blood-brain barrier function   - Hemichannels contribute to Ca2+ dynamics in smooth muscle cells in acutely isolated small mesenteric arteries   - Cell death communication via connexin channels investigated in a C6 glioma cell model   - Inhibition of connexin 43 hemichannel responses with high cytoplasmic calcium concentrations is mediated by mechanisms different from calcium activation of hemichannel responses   - Calcium regulation of connexin-43 hemichannel-mediated ATP release in glial cells De Bundel D. - Anticonvulsive effects of insulin-regulated aminopeptidase deletion in a pentylenetetrazol mouse seizure model are not mediated by the accumulation of somatostatin-14 De Herdt V. - In vivo measurements of vagus nerve compound action potentials in rats   - Clinical added value of meg in patients with refractory partial epilepsy and non-localizing conventional presurgical evaluation De Meyer G.R.Y. - Lithium chloride induces macrophage apoptosis in rabbit atherosclerotic plaques De Meyer I. - Lithium chloride induces macrophage apoptosis in rabbit atherosclerotic plaques De Tiege X. - Clinical added value of meg in patients with refractory partial epilepsy and non-localizing conventional presurgical evaluation De Vuyst E. - Connexin channels contribute to endothelial Ca2+ dynamics and alter blood-brain barrier function   - Calcium regulation of connexin-43 hemichannel-mediated ATP release in glial cells   - Inhibition of connexin 43 hemichannel responses with high cytoplasmic calcium concentrations is mediated by mechanisms different from calcium activation of hemichannel responses   - Cell death communication via connexin channels investigated in a C6 glioma cell model   - Hemichannels contribute to Ca2+ dynamics in smooth muscle cells in acutely isolated small mesenteric arteries Deblaere K. - Clinical added value of meg in patients with refractory partial epilepsy and non-localizing conventional presurgical evaluation Decleves A.E. - Analysis of renal phenotype of hyaluronidase-1 or hyaluronidase-2 knockout mice   - Temporal evolution of inflammation and oxidative stress in the rat post-ischemic kidney Decrock E. - Connexin channels contribute to endothelial Ca2+ dynamics and alter blood-brain barrier function   - Hemichannels contribute to Ca2+ dynamics in smooth muscle cells in acutely isolated small mesenteric arteries   - Cell death communication via connexin channels investigated in a C6 glioma cell model   - Calcium regulation of connexin-43 hemichannel-mediated ATP release in glial cells   - Inhibition of connexin 43 hemichannel responses with high cytoplasmic calcium concentrations is mediated by mechanisms different from calcium activation of hemichannel responses DeDeken X. - Expression and localization of Na+-Hco3- cotransporter (NBCe1) in thyroid and its regulation by TSH Delbeke J. - In vivo measurements of vagus nerve compound action potentials in rats Deleuze S. - Body size affects morphological echocardiographic parameters in the equine species Deleye S. - Evaluation of hippocampal deep brain stimulation by SPECT Demaegdt H. - Anticonvulsive effects of insulin-regulated aminopeptidase deletion in a pentylenetetrazol mouse seizure model are not mediated by the accumulation of somatostatin-14   - [3H]AL-11, A new stable and selective ligand for the IRAP/AT4 receptor in both membranes and intact cells Dilly S. - Combined experimental and computational approaches to study the action of blockers of small conductance calcium-activated potassium (SK) channels Dumont J.E. - Expression and localization of Na+-Hco3- cotransporter (NBCe1) in thyroid and its regulation by TSH