Objective: The year 2010 is the 100th anniversary of the publication in Acta Physiologica of the Seven Little Devils, the papers by August and Marie Krogh that sought to demolish the theory of Christian Bohr that oxygen diffusion from the lungs to the circulation is facilitated by a specific cellular activity. The bone of contention was the transport of oxygen from the lungs into the bloodstream: Are passive transport and diffusion capacity together high enough to secure the oxygen supply in all circumstances, or is there an additional specific ("active") cell activity responsible for the transport of oxygen. The purpose of this reexamination is to determine whether the arguments of the contestants lead to the same conclusion today that observers reached in the years after 1910. Methods: Reexamination of the papers published by the contestants reveals that it is unlikely that most observers read the papers of both contestants sufficiently well in the original languages to reach a fair conclusion on the state of controversy. In particular, it appears that observers failed to read Bohr's papers with the necessary care. These sins of omission are now rectified. Results: Reexamination of the Seven Little Devils and Bohr's major response (1909) shows that the contestants' views were more consistent than the contestants themselves or posterity have recognized. Posterity judged the dispute from the victor's perspective, but it is a fact that August Krogh's Nobel Prize was awarded for the discovery of the "specific" cellular activity, now known as capillary recruitment, that Bohr originally introduced to explain the discrepancy between diffusion capacities at rest and work. Conclusion: Christian Bohr was correct in the broader sense of a cellular contribution to diffusion capacity: The diffusion capacity at rest is not high enough to explain the transport during work; a special mechanism intervenes and optimizes the conditions under which diffusion acts. August Krogh, on the other hand, was right in the narrower sense that the diffusion mechanism remains entirely passive.